| DEBATE
Legalisation of the remarkably safe cannabis plant would require
no special regulations
Date: Mon, 26 Jun 2000 18:20:46 +0100 From: "WebBooks.org.uk"
<clcia@paston.co.uk>
To: ukcia-l@mimir.com
Claims of various degrees of harmfulness and danger associated with
the smoking of cannabis are often cited by prohibitionists and those
who favour regulated legalisation alike.
Whilst many people agree that prohibition is unjust and ineffective,
they differ in their opinions on the needs for regulations. Whilst
few would want unnecessary regulations and limitations legislated
onto cannabis once legal, few also would want to see no regulations
installed if indeed necessary. It is therefore vital that we attempt
to reach some sort of conclusion on the harm or potential harm through
individual or widespread cannabis use.
I have personally read many reports from scientific and empirical
studies on actual cannabis use, as well as some of those based upon
laboratory tests carried out on mice, rats, rabbits and monkeys,
using concentrated and synthetic THC - tetrahydrocannabinol - one
of the main active ingredients found in the parts of the cannabis
plant used recreationally and medically, particularly the tops and
heads.
My own studies of the evidence from both sides has led me to the
following conclusions:
1. All of the allegations of harm are based upon dubious work, laboratory
experiments not involving cannabis and not involving tests on humans,
and unreliable anecdote often exaggerated and g by drug workers.
2. Cannabis is indeed "remarkably safe" and free from
danger, barring of course the obvious dangers of being hit over
the head with a large lump of resin.
"We say that on the medical evidence available, moderate indulgence
in cannabis has little ill-effect on health, and that decisions
to ban or legalise cannabis should be based on other considerations.":
The Lancet, vol 352, number 9140, November 14 1998
As it is nonsensical to attempt to prove any substance to be completely
harmless under all circumstances, I am tackling this issue by listing
the various harm allegations and counteracting them with quotes
from and references to the experts.
harm allegations
* #Myth: Cannabis is toxic / poisonous
* #Myth: Cannabis intoxicates
* #Myth: Cannabis is addictive
* #Myth: Cannabis causes hallucinations
* #Myth: Cannabis causes cancer
* #Myth: Cannabis smoking damages the lungs
* #Myth: Cannabis suppresses the immune system.
* #Myth: Cannabis causes impotency / infertility
* #Myth: Cannabis destroys short-term memory
* #Myth: Cannabis detrimentally effects motor co-ordination / driving
skill
* #Myth: Cannabis detrimentally effects cognitive
* #Myth: Cannabis causes a-motivation / laziness
* #Myth: Cannabis use leads to the use of hard drugs
Myth: Cannabis is toxic/poisonous From: OPINION AND RECOMMENDED
RULING, FINDINGS OF FACT, CONCLUSIONS OF LAW AND DECISION OF Administrative
LAW JUDGE, DATED: SEP 6 1988
http://www.paston.co.uk/users/webbooks/young88.html
Findings of Fact:
* "4. Nearly all medicines have toxic, potentially lethal effects.
But marijuana is not such a substance. There is no record in the
extensive medical literature describing a proven, documented cannabis-induced
fatality.
* "5. This is a remarkable statement. First, the record on
marijuana encompasses 5,000 years of human experience. Second, marijuana
is now used daily by enormous numbers of people throughout the world.
Estimates suggest that from twenty million to fifty million Americans
routinely, albeit illegally, smoke marijuana without the benefit
of direct medical supervision. Yet, despite this long history of
use and the extraordinarily high numbers of social smokers, there
are simply no credible medical reports to suggest that consuming
marijuana has caused a single death.
* "6. By contrast aspirin, a commonly used, over-the-counter
medicine, causes hundreds of deaths each year.
* "7. Drugs used in medicine are routinely given what is called
an LD-50. The LD-50 rating indicates at what dosage fifty percent
of test animals receiving a drug will die as a result of drug induced
toxicity. A number of researchers have attempted to determine marijuana's
LD-50 rating in test animals, without success. Simply stated, researchers
have been unable to give animals enough marijuana to induce death.
* "8. At present it is estimated that marijuana's LD-50 is
around 1:20,000 or 1:40,000. In layman terms this means that in
order to induce death a marijuana smoker would have to consume 20,000
to 40,000 times as much marijuana as is contained in one marijuana
cigarette. NIDA-supplied marijuana cigarettes weigh approximately
.9 grams. A smoker would theoretically have to consume nearly 1,500
pounds of marijuana within about fifteen minutes to induce a lethal
response.
* "9. In practical terms, marijuana cannot induce a lethal
response as a result of drug-related toxicity.
* "10. Another common medical way to determine drug safety
is called the therapeutic ratio. This ratio defines the difference
between a therapeutically effective dose and a dose which is capable
of inducing adverse effects.
* "11. A commonly used over-the-counter product like aspirin
has a therapeutic ratio of around 1:20. Two aspirins are the recommended
dose for adult patients. Twenty times this dose, forty aspirins,
may cause a lethal reaction in some patients, and will almost certainly
cause gross injury to the digestive system, including extensive
internal bleeding.
* "12. The therapeutic ratio for prescribed drugs is commonly
around 1:10 or lower. Valium, a commonly used prescriptive drug,
may cause very serious biological damage if patients use ten times
the recommended (therapeutic) dose.
* "13. There are, of course, prescriptive drugs which have
much lower therapeutic ratios. Many of the drugs used to treat patients
with cancer, glaucoma and multiple sclerosis are highly toxic. The
therapeutic ratio of some of the drugs used in antineoplastic therapies,
for example, are regarded as extremely toxic poisons with therapeutic
ratios that may fall below 1:1.5. These drugs also have very low
LD-50 ratios and can result in toxic, even lethal reactions, while
being properly employed.
* "14. By contrast, marijuana's therapeutic ratio, like its
LD-50, is impossible to quantify because it is so high." In
the journal FUNDAMENTAL AND APPLIED TOXICOLOGY, Dr. William Slikker,
director of the Neurotoxicology Division of the National Center
for Toxicological Research (NCTR), described the health of monkeys
exposed to very high levels of cannabis for an extended period:
"The general health of the monkeys was not compromised by a
year of marijuana exposure as indicated by weight gain, carboxyhemoglobin
and clinical chemistry/hematology values."
(TOXICOLOGY LETTERS, No Increase in Carcinogen-DNA Adducts in the
Lungs of Monkeys Exposed Chronically to Marijuana Smoke, 1992, Dec;63
(3): 321-32.
THE ARKANSAS TIMES (Refer Madness. 16 Sept 1993) asked Dr. Merle
Paule of NCTR about evidence of cannabis toxicity and the health
of the monkeys in the study, Dr. Paule said,
"There's just nothing there. They were all fine."
Myth: Cannabis intoxicates
This is really a matter of semantics, as, strictly speaking, a non-toxic
substance cannot 'intoxicate'. "intoxication" is usually
and often detectable simply by a detrimental effect upon motor and
cognitive skills; these are covered below.
Myth: Cannabis is addictive
Here we must distinguish between firstly, addictiveness and dependency,
and secondly, between medical and psychological dependency.
Medical dependency is not really the issue here, since it is perfectly
natural and acceptable for a person to be dependent upon a medicine
to ease their suffering, given that the medicine is at least reasonably
and acceptably safe.
TRENDS IN PHARMACOLOGICAL SCIENCES: Neurobiology of Marijuana Abuse.
1992, 13:201-206. pg. 203:
"research shows cannabis has limited potential for development
of...psychological dependence due to the weak reinforcing properties
of Delta-9-THC."
BRAIN RESEARCH JOURNAL: Chronic cannabinoid administration alters
cannabinoid receptor binding in rat brain: a quantitative
autoradiographic study. 1993, 616:293-302. pg. 300.
"cannabinoid dependence and withdrawal phenomena are minimal."
The Shafer Commission (USA) of 1970 said:
"Marijuana does not lead to physical dependency, although some
evidence indicates that the heavy, long-term users may develop a
psychological dependence on the drug”
The Panama Canal Zone Military Investigations (US Military, 1929)
said:
"There is no evidence that Marihuana as grown and used [in
the Canal Zone] is a 'habit-forming' drug."
In 1997, (R. v Clay), Ontario Justice John McCart (Canada) ruled,
"Cannabis is not an addictive substance."
B.C. Justice F.E. Howard in a similar case confirmed his findings
in 1998.
US Department of Health and Human Services, 1991:
"Given the large population of marijuana users and the infrequent
reports of medical problems from stopping use, tolerance and dependence
are not major issue at present."
("Drug Abuse and Drug Abuse Research, Rockville, MD, (1991)
p C3
Myth: Cannabis causes hallucinations
Report of the Australian Government, 1992:
"Cannabis has been erroneously classified as a narcotic, as
a sedative and most recently as an hallucinogen. While the cannabinoids
do possess hallucinogenic properties, together with stimulant and
sedative effects, they in fact represent a unique pharmacological
class of compounds. Unlike many other drugs of abuse, cannabis acts
upon specific receptors in the brain and periphery.The discovery
of the receptors and the naturally occurring substances in the brain
that bind to these receptors is of great importance, in that it
signifies an entirely new pathway system in the brain."
Myth: Cannabis causes cancer
BOSTON, Jan. 30, 1997 (UPI): "The U.S. federal government has
failed to make public its own 1994 study that undercuts its position
that marijuana is carcinogenic - a $2 million study by the National
Toxicology Program. The program's deputy director, John Bucher (http://www.niehs.nih.gov/dirtob/bucher.htm),
says the study "found absolutely no evidence of cancer."
In fact, animals that received THC had fewer cancers. Bucher denies
his agency had been pressured to shelve the report, saying the delay
in making it public was due to a personnel shortage.
CANCER PREVENTION DATA "Marijuana Use and Mortality":
AMERICAN JOURNAL OF PUBLIC HEALTH,
April 1997: TABLE 2 Relative Risk of Death for Ever Users and Current
Users of Marijuana, by Sex and Cause of Death: Kaiser Pemanente
Medical Care Program Members (n = 65,171), Oakland and San Francisco,
June 1979 through December 1985 - section of table regarding cancer
(Neoplasms) as the cause of death:
MEN Ever Users Relative Risk of Cancer Death Full Model 0.78 Non-smokers/
Occasional Drinkers 0.46 Current Users Full Model 0.97 Non-smokers/
Occasional Drinkers 0.75
WOMEN Ever Users Full Model 0.82 Non-smokers/ Occasional Drinkers
0.70 Current Users Full Model 0.86 Non-smokers/ Occasional Drinkers
0.56
Here, numbers less than one for Relative Risk of Cancer Death represent
a lower rate of fatal cancer among marijuana smokers in the large
Kaiser Study from California. For example, women who are current
marijuana smokers but did not smoke tobacco were found to have only
56% of the risk of cancer death as compared to other women who were
non-smokers of both tobacco and marijuana.
Not only is the evidence linking cannabis smoking to cancer negative,
but the largest human studies cited indicated that cannabis users
had lower rates of cancer than nonusers. What's more, those who
smoked both cannabis and tobacco had lower rates of lung cancer
than those who smoked only tobacco-a strong indication of chemoprevention.
Even more, in 1975 researchers at the Medical College of Virginia
found that cannabis showed powerful antitumour activity against
both benign and malignant tumours (the government then banned all
future cannabis/cancer research).
(The Emperor Wears No Cloths. Jack Herer, Queen of Clubs Pub, 1991)
(Ganja in Jamaica: A Medical Anthropological Study of Chronic Marijuana
Use. 1975. Anchor Books)
(Cannabis in Costa Rica: A Study of Chronic Marijuana Use, 1980-82,
Institute for the Study of Human Issues, 3401 Science Center Philadelphia,
PA.)
The NEW ENGLISH DISPENSATORY of 1764 recommends boiled cannabis
roots for the elimination of tumours.
(Marijuana: The First 12,000 Years. PlenumPress, 1980)
Powerful evidence that cannabis not only does not cause cancer,
but that it may prevent and even cure cancer:
<http://www.erowid.org/plants/cannabis/cannabis_health2.shtml>
SO, YOU THOUGHT IT WAS THE TAR THAT CAUSED CANCER
Myth: Cannabis smoking damages the lungs
Researchers at the University of California (UCLA) School of Medicine
have announced the results of an 8 - year study into the effects
of long-term cannabis smoking on the lungs. In Volume 155 of the
American Journal of Respiratory and Critical Care Medicine, Dr.
D.P. Tashkin reported
“Findings from the present long-term, follow-up study of heavy,
habitual marijuana smokers argue against the concept that continuing
heavy use of marijuana is a significant risk factor for the development
of [chronic lung disease. ..Neither the continuing nor the intermittent
marijuana smokers exhibited any significantly different rates of
decline in [lung function]" as compared with those individuals
who never smoked marijuana. Researchers added:
"No differences were noted between even quite heavy marijuana
smoking and non-smoking of marijuana."
Myth: Cannabis suppresses the immune system.
Two studies in 1978 and 1988 showed that cannabis actually stimulated
the immune system
From: "Exposing Marijuana Myths:(The Lindesmith Center)"
"False: Marijuana Impairs Immune System Functioning" It
has been widely claimed that marijuana substantially increases users'
risk of contracting various infectious diseases. First emerging
in the 1970s, this claim took on new significance in the 1980s,
following reports of marijuana use by people suffering from AIDS.
"THE FACTS"
The principal study fueling the original claim of immune impairment
involved preparations created with white blood cells that had been
removed from marijuana smokers and controls. After exposing the
cells to known immune activators, researchers reported a lower rate
of transformation in those taken from marijuana smokers. "However,
numerous groups of scientists, using similar techniques, have failed
to confirm this original study. "In fact, a 1988 study demonstrated
an increase in responsiveness when white blood cells from marijuana
smokers were exposed to immunological activators.
"Studies involving laboratory animals have shown immune impairment
following administration of THC, but only with the use of extremely
high doses. For example, one study demonstrated an increase in herpes
infection in rodents given doses of 100 mg/kg/day -- a dose approximately
1000 times the dose necessary to produce a psychoactive effect in
humans.
"There have been no clinical or epidemiological studies showing
an increase in bacterial, viral, or parasitic infection among human
marijuana users.
In three large field studies conducted in the 1970s, in Jamaica,
Costa Rica and Greece, researchers found no differences in disease
susceptibility between marijuana users and matched controls.
"Marijuana use does not increase the risk of HIV infection;
nor does it increase the onset or intensity of symptoms among AIDS
patients. In fact, the FDA decision to approve the use of Marinol
(synthetic THC) for use in HIV-wasting syndrome relied upon the
absence of any immunopathology due to THC."
Today, thousands of people with AIDS are smoking marijuana daily
to combat nausea and increase appetite. There is no scientific basis
for claims that this practice compromises their immune responses.
Indeed, the recent discovery of a peripheral cannabinoid receptor
associated with lymphatic tissue should encourage aggressive exploration
of THC's potential use as an immune-system stimulant.
"From also Marijuana Myths, Marijuana Facts": Lynn Zimmer
Ph.D. and John P. Morgan M.D. "
At the 1981 conference on marijuana sponsored by the World Health
Organisation and Canada's Addiction Research Foundation, reviewers
of the research literature on immunity reported
"There is no conclusive evidence that cannabis predisposes
man to immune dysfunction".
A few years later, in approving THC (Marinol) for use as a medicine,
the FDA found no convincing evidence that THC caused immune impairment.
In 1992, the FDA approved Marinol as an appetite stimulant specifically
for AIDS patients, who have serious immunosuppression.
"Marijuana Myths, Marijuana Facts": Lynn Zimmer Ph.D.
and John P. Morgan M.D. ISBN 0-9641568-4-9; page 107.Munson and
Fehr (1983) note 15, page 338 Food and Drug Administration, "Unimed's
Marinol (Dronabinol)Lau, R.J. et al "Phytohemagglutinin-Induced
Lymphocyre Transformations in Humans Receiving Delta-9-Tetrahydrocannabinol,"
Science 192, 805-07 (1976)Dax, EM. Et al., "The Effects of
9_ENE-Tetrahydrcannabinol on Hormone Release and Immune Function,"
Journal of Steroid Biochemistry 34: 263-70 (1989)
Myth: Cannabis causes impotency / infertility
From: "Exposing Marijuana Myths: (The Lindesmith Center)"
page 93;
"Studies of men in the general population have also failed
to find differences in the testosterone levels of marijuana users
and nonusers.""There is no convincing evidence of infertility
related to marijuana consumption in humans.""There are
no epidemiological studies showing that men who use marijuana have
higher rates of infertility than men who do not. Nor is there evidence
of diminished reproductive capacity among men in countries where
marijuana use is common." Abel, E.L., et al, "Marijuana
and Sex: A Critical Survey," Drug and Alcohol Dependence 8:
1-22 (1981)
Ehrenkranz, J.R.L. and Hembee, WC., "Effects of Marijuana on
Male Reproductive Function," Psychiatric Annals 16: 243-49
(1986)
Cushman, P, "Plasma Testosterone Levels in Healthy Male Marijuana
Smokers," American Journal of Drug and Alcohol Abuse 2: 269-75
(1975)
Block, R I, et al, "Effects of Chronic Marijuana Use of Testosterone,
Luteinizing Hormone, Follicle Stimulating Hormone, Prolactin and
Cortisol in Men and women,"Drug and Alcohol Dependence 28,:
121-28 (1991)
Myth: Cannabis destroys short-term memory
The Australian Government Report 1996:
"The weight of the available evidence suggests that the long-term
heavy use of cannabis does not produce any severe impairment of
cognitive function."
Myth: Cannabis detrimentally effects motor co-ordination / driving
skill
Crancer Study, Washington Department of Motor Vehicles:
“Simulated driving scores for subjects experiencing a normal
social ‘high’ and the same subjects under control conditions
are not significantly different. However, there are significantly
more errors for alcohol intoxicated than for control subjects”
U.S. Department of Transportation, National Highway Traffic Safety
Administration (DOT HS 808 078), Final Report, November 1993:
"THC's adverse effects on driving performance appear relatively
small"
Sutton (1983) also found that cannabis had little effect on actual
driving performance.
"Driving in traffic, however, while showing a trend toward
poorer performance, was not significantly affected, and the effects
of cannabis were much more variable."
The Australian Government Report, 1996, page 6;
"There is no controlled epidemiological evidence that cannabis
users are at increased risk of being involved in motor vehicle or
other accidents.
Myth: Cannabis detrimentally effects cognitive skills US: Cannabis
Use and Cognitive Decline in Persons under 65 Years of Age Pubdate:
1 May 1999
Source: American Journal of Epidemiology Copyright: 1999 Johns Hopkins
University School of Hygiene and Public Health Ref: Am J Epidemiol
1999; 149:794-800 Mail: 111 Market Place, Suite 840, Baltimore MD
21202 U.S.A. <http://www.jhsph.edu/Publications/JEPI/">
Website Authors: Constantine G. Lyketsos, Elizabeth Garrett, Kung-Yee
Liang, and James C. Anthony (Osler 320, The Johns Hopkins Hospital,
600 North Wolfe Street, Baltimore, MD 21287-5371)
"The purpose of this study was to investigate possible adverse
effects of cannabis use on cognitive decline after 12 years in persons
under age 65 years. This was a follow-up study of a probability
sample of the adult household residents of East Baltimore. The analyses
included 1,318
participants in the Baltimore, Maryland, portion of the Epidemiologic
Catchment Area study who completed the Mini-Mental State (MMSE)
examination during three study waves in 1981, 1982, and 1993--1996.
Individual MMSE score differences between waves 2 and 3 were calculated
for each study participant. After 12 years, study participants'
scores declined a mean of 1.20 points on the MMSE (standard deviation
1.90), with 66% having scores that declined by at least one point.Significant
numbers of scores declined by three points or more (15% of participants
in the 18--29 age group).
* There were no significant differences in cognitive decline between
heavy users, light users, and nonusers of cannabis.
* There were also no male-female differences in cognitive decline
in relation to cannabis use.
The authors conclude that over long time periods, in persons under
age 65 years, cognitive decline occurs in all age groups This decline
is closely associated with ageing and educational level but does
not appear to be associated"
Ethiopian Zion Coptic Church Study, 1980
"Some participants had smoked at least two to four large cigarettes
(each containing 1/4 to 1/2 ounce of cannabis) over 16 hours a day
for periods of up to 50 years. "...the most impressive thing...
is the true paucity of neurological abnormalities.
Schaeffer: A Neuropsychological Evaluation; A Case History ...I.Q.’s
of Zion Coptics increased after they began to use ganga;
"Heavy cannabis consumers suffered no apparent psychological
or physical harm."
US Jamaican Study 1974:
"No impairment of physiological, sensory and perceptual performance,
tests of concept formation, abstracting ability, and cognitive style,
and tests of memory"
Myth: Cannabis causes a-motivation / lazinessWe must of course distinguish
between those people who are naturally or by habit or psychological
so set as lazy or a-motivated, and any such a-motivation caused
by cannabis consumption.
Dr. Andrew Weil (Rubin & Comitas Ganja in Jamaica, 1975) said
“a-motivation [is] a cause of heavy marijuana smoking rather
than the reverse”
In 1997, (R. v Clay), Ontario Justice John McCart (Canada) ruled,
"Cannabis … does not cause a motivational syndrome."
His findings were confirmed by B.C. Justice F.E. Howard in a similar
case in 1998
Myth: Cannabis use leads to the use of hard drugsConsidering the
millions of people in the UK, and the hundreds of millions around
the world, who have used cannabis for short or long periods, it
is clear that if it led to the use of hard and addictive drugs there
would be many more new addicts that we have seen.
We must, here, also remember that under the UK and other government
policies of "tackling drugs together", under a regime
that prohibits hard drugs alongside cannabis, where the supplies
remain in criminal control, it is often the case that people may
be led from one substance to another by their peers and by their
suppliers. This does not of course mean that cannabis itself is
a gateway or hard drug use.
We must also remember that at least a proportion of cannabis users
may be people prone to trying other substances, whether by way of
n, research, 'spiritual' quest, or psychological imbalance.
The LaGuardia sub-committee of New York 1944 said: “The use
of marijuana does not lead to morphine or heroin or cocaine addiction
and no effort is made to create a market for these narcotics by
stimulating the practice of marijuana smoking”
"Marijuana: Facts for Teens." U.S. Department of Health
and Human Services. Washington, D.C. 1995, p.10.: "Most marijuana
users do not go on to use other drugs." :
Jack Straw, The Daily Telegraph, 3 April 2000: "While it is
undoubtedly the case that many drug addicts started with cannabis,
to claim that taking cannabis is bound to lead to hard drugs has
always seemed to me far-fetched."
Drugs Policy in the Netherlands (1995): Dutch Ministry of Health,
Welfare and Sport
"Moreover, users of soft drugs do not as a rule tend to experiment
with hard drugs, such as heroin or cocaine; this is indeed the intention
of the policy of keeping the markets separate. There is little use
of heroin and cocaine among minors in the Netherlands, and the trend
is towards even less."Myth: Increased availability will lead
to increased usage"
Drugs Policy in the Netherlands (1995): Dutch Ministry of Health,
Welfare and Sport"4.1. Extent and nature of cannabis use "The
decriminalisation of the possession of soft drugs in 1976 did not
result in increased use. The level of consumption stabilised in
the first few years after the Opium Act was amended. According to
national figures, use again increased somewhat between 1984 and
1994, a trend which has also been observed elsewhere. Indeed, the
United States has experienced a considerable increase in recent
years. "Both as regards the extent of cannabis use and trends
in use, the Netherlands differs very little from other countries.
"As already indicated, the number of users of soft drugs has
increased after falling in the 1970s. Patterns of consumption are
overwhelmingly recreational, though among certain specific categories
of young people, such as chronic truants and street children, the
use of cannabis can be described as very substantial and intensive.
"The policy pursued by the Netherlands does not appear to have
led to an i ncrease in use, though there are indications that the
existence of freely accessible coffee shops means that certain users
continue to use the drugs for longer. "Conclusions and policy
intentions "The decriminalisation of the possession of quantities
of soft drugs for personal use and the existence of sales points
tolerated under certain circumstances by the authorities have not
resulted in a worryingly high level of consumption among young people.
Moreover, users of soft drugs do not as a rule tend to experiment
with hard drugs, such as heroin or cocaine; this is indeed the intention
of the policy of keeping the markets separate.
There is little use of heroin and cocaine among minors in the Netherlands,
and the trend is towards even less."The effects of partial
decriminalisation on cannabis use in South Australia, 1985 to 1993
National Drug and Alcohol Research Centre, University of New South
Wales, Sydney Aust J Public Health, 19: 3, 1995 Jun, 281-7:"
In 1987 the Cannabis Expiration Notice scheme decreased penalties
for the personal use of cannabis in South Australia. Data from four
National
Campaign Against Drug Abuse (NCADA) household drug-use surveys covering
the period 1985 to 1993 were analysed to measure the effect of the
decriminalisation on cannabis use. The main outcomes used were the
self-reported prevalence rates of having ever used cannabis and
current
weekly use. Logistic regression was used to control for the potentially
confounding effects of age and sex. Other outcomes were rates of
having ever been offered cannabis and willingness to use cannabis
if offered it. Between 1985 and 1993 the adjusted prevalence rate
of ever having used cannabis increased in South Australia from 26
per cent to 38 per cent. There were also significant increases in
Victoria and Tasmania, and to a lesser extent in New South Wales.
The increase in South Australia was not significantly greater than
the average increase (P = 0.1). Adjusted rates of weekly use increased
between 1988 and 1991 in South Australia, but did not change through
1993. Although the effect was in the direction of a greater increase
in South Australia, this was not statistically significant when
compared to increases in the rest of Australia (P = 0.07). The greatest
increase in adjusted weekly use occurred in Tasmania between 1991
and 1993, from 2 per cent to 7 per cent. Although the NCADA survey
data indicate that there were increases in cannabis use in South
Australia in 1985-1993, they cannot be attributed to the effects
of partial decriminalisation, because similar increases occurred
in other states"
And now, some general quotes on the health effects of smoking cannabis:
March 20, 1997, Sydney, Australia: The National Drug and Alcohol
Research Centre in Australia. The study, which involved interviews
with 268 marijuana smokers and 31 non-using partners and family
members, is one of the first ever conducted in Australia to determine
the effects of
long-term marijuana use. Its findings were reported by the Sydney
Morning Herald last month. "We don't see evidence of high psychological
disturbance among the [long- term users,]" said chief investigator
David Reilly. "The results seem unremarkable; the exceptional
thing is that the respondents are unexceptional."
The Report of the Australian Government 1996 says:
“The ... major possible adverse effects of chronic, heavy
cannabis use ... remain to be confirmed”
“The major health and psychological effects of chronic cannabis
use, especially daily use over many years, remain uncertain”
“As has been stressed ... there is uncertainty. ......To varying
degrees....inferences from animal research, laboratory studies,
and clinical observations about probable ill effects. In some cases
inferences depend upon arguments from what is known about the adverse
effects of other drugs, such as tobacco and alcohol”.
“The probable and possible adverse health and psychological
effects of cannabis need to be placed in comparative perspective
to be fully appreciated”.The USA Merck Manual of Diagnosis
and Therapy (1987) says: “Cannabis can be used on an episodic
but continual basis without evidence of social or psychic dysfunction.
In many users the term dependence with its obvious connotations,
probably is mis-applied... The chief opposition
to the drug rests on a moral and political, and not toxicologic,
foundation”.
Jamaican Study 1970
:“... as a multipurpose plant, ganga is used medicinally,
even by non-smokers. ....There were no indications of organic brain
damage or chromosome damage among smokers and no significant clinical
psychiatric, psychological or medical) differences between smokers
and controls.”
UK Royal Commission, The Wootton Report, UK, 1968:
“Having reviewed all the material available to us we find
ourselves in agreement with the conclusion reached by the Indian
Hemp Drugs Commission appointed by the Government of India (1893-94)
and the New York Mayor’s Committee (1944 - LaGuardia) that
the long-term consumption of cannabis in moderate doses has no harmful
effects”LaGuardia Commission Report, 1944
"Cannabis [smoking] does not lead directly to mental or physical
deterioration... Those who have consumed marijuana for a period
of years showed no mental or physical deterioration which may be
attributed to the drug"Panama Canal Zone Report, 1925
"There is no evidence... that any deleterious influence on
the individual using [cannabis]"
Indian Hemp Drugs Commission, 1894
"The commission has come to the conclusion that the moderate
use of hemp drugs is practically attended by no evil results at
all. … ...moderate use of hemp... appears to cause no appreciable
physical injury of any kind,... no injurious effects on the mind...
[and] no moral injury whatever."
see also:
http://www.paston.co.uk/users/webboks/serious-crime.html
http://www.paston.co.uk/users/webbooks/goddard.html
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